How green is your medicine?

I like this quote from Genie Rowson:

Green medicine stimulates the body's own healing mechanisms to repair and rebuild itself, and help reverse illness and encourage healing. This approach differs considerably from the more conventional medicine we know so well, which suppresses our symptoms (which are actually the body's healing mechanisms). But does green medicine really work? The answer is yes. Absolutely! Greener systems of medicine such as acupuncture, Chinese medicine and homeopathy are gaining new converts every day, because they really do work! Millions of people worldwide have been healed through these gentler, safer, non-toxic alternatives. There is a wordwide movement towards greener options in every aspect of life.

If you're into homeopathy, check out Genie's web site - There's A Remedy For That. She has a great ailments page with suggestions for homeopathic remedies for a whiole host of complaints.

Archives July 2009

July 30My debut in Walima Club :Egyptian Special - Koshary and Basbousa(Semolina cake)

July 28Kosambari,a raw vegetable and lentil salad

July 27Daring Bakers...Milan cookies


July 23Dodol(Coconut Milk Jaggery sweet)

July 21Eggcelent Eggplant recipes :Ratatouille Pasta,Kashmiri Baigan and Stuffed Baby eggplants

July 20Bisi Bele Bath(Hot Lentil Rice)

July 15Ice Dream Cook Book Review.Roasted Banana nut Ice Dream.Peach Melba

July 11Shahi Paneer(Indian Cottage Cheese)

July 8Beet Chalupas


July 6Rosemary Zopf(Swiss Braided Bred)

July 6Grilled Vegetable Wild Rice Salad

July 216 Bean Stew With Whole Grain Dinner Rolls

July 1I Love Cilantro!:Cilantro Coconut Milk Pasta

Get fit to lower your colon cancer risk

The Facts:
* Approximately 200 patients who had undergone a colonoscopy in the previous 3 months either continued their usual low exercise lifestyle or started a 12-month exercise programme, in a study to investigate the link between exercise and colon cancer.
* Cells from the colon (lower bowel) were examined at the start and end of the study, for specific markers that are associated with colon cancer.
* Male patients, who had performed on average at least 250 minutes of exercise a week, were found to have a lower level of cancer markers than patients with a lower level or no exercise. No effect was seen in women.

Shaun’s comment: In this large study, strenuous exercise resulted in changes in the bowel cells that would be likely to reduce the chance of cancer. Some caution is required, as the researchers did not actually prove that it reduced bowel cancer. But they did provide some evidence and, importantly, a mechanism to explain how exercise could reduce bowel cancer risk. Overall, a fascinating study and one more great reason to buy some decent running shoes.

Study reference:
http://cebp.aacrjournals.org/cgi/content/abstract/15/9/1588

Comment on eHealth exchange privacy policies

eHealthConnecticut has created a portal to collect public comment on evolving privacy and security policies for CT’s health information exchange. Posted so far is the draft Universal Medical Records Release form that patients will sign to indicate if they agree to share their information and materials from our two input forums held at the Capitol. Coming soon will be privacy and security policies that will govern the exchange. You can comment with attribution or anonymously and sign up to receive alerts when new documents are posted.
Ellen Andrews

CVE meeting in DC

I’ve spent the last two days at a fascinating meeting of data geeks, providers, employers, health plans, quality organizations and consumer advocates from across the country all working to improve the quality of health care. It was a meeting of Chartered Value Exchanges from 25 communities/states; eHealthConnecticut was designated as a CVE in a very competitive process. The program is administered by the federal Agency for Health Care Research and Quality (AHRQ), part of Health and Human Services. The best part of the conference is the strong emphasis and clear commitment of AHRQ and the CVEs to serving consumers’ needs. The first sentence on the website sums it up -- “Consumers deserve to know the quality and cost of their health care.” Everything flows from there. It isn’t just a set of platitudes, but it permeates everything they do. It is refreshing – consumer advocates in CT don’t get that a lot.

Eventually, through eHealthConnecticut CT consumers will be able to get comparative quality information about our providers across payers online– for example how many patients with diabetes are being well managed, and STD or cancer screening rates. In a few communities, this ability already exists. A great example is the Puget Sound Health Alliances’ Community Check up. They also have a special report comparing care in Medicaid with other coverage. There was a lot of talk about fair, nationally certified, consensus approved measures and standards, as well as lots and lots of discussion and research about how best to meet consumers’ needs and questions, how to present the information to ensure it is useful, and how to market the tools. CVEs are devoting significant resources to answering all these questions – making sure the system is consumer-centered. It is a fantastic model that CT policymakers should be studying.

Another great benefit from the conference was getting to spend two days with the rest of the CT delegation, including a health plan, a large employer, a primary care physician, a hospital rep., and a quality organization. It is critical for stakeholders to understand and respect other perspectives, and the opportunities for that in CT are rare, unfortunately.
Ellen Andrews

Back again

I’ve taken my blog back, but am on the road again and have decided to take a break this week from blogging. This would be a great time for those of you who weren’t able to keep up with our blog posts from D.C. last week to look them over now. Blog: Hijacked!Day 1: Disaster Preparedness, Health Care Reform and Health Care IT (written by Abby Lowe)Video: One stop on our whirlwind tourDay 2:

Back again

I’ve taken my blog back, but am on the road again and have decided to take a break this week from blogging. This would be a great time for those of you who weren’t able to keep up with our blog posts from D.C. last week to look them over now. Blog: Hijacked!Day 1: Disaster Preparedness, Health Care Reform and Health Care IT (written by Abby Lowe)Video: One stop on our whirlwind tourDay 2:

My Debut in Walima Cooking Club :Egyptian Special - Koshary and Basbousa(Semolina cake)

In Arlette's words-
"The Arabic word Walima (banquet) is derived from the root word Walam, which literally means to gather and assemble. The Arabs used it for a meal or feast where people were invited and gathered to eat and celebrate for days, either for new born babies, a traveler returning home or for weddings.Later the term became exclusive for banquet celebration."
(The Logo is from one of Lebanese famous Painter, Mustapha Faroukh’s “Still Life Album".)

Walima cooking club celebrates Middle Eastern food.Each month foodies(members of the club) around the world cook a specialty dish of the region.The July 2009, Challenge was choosen by Talented Chahira from Chahira's Cuisine.She choose 2 prominent dishes of Egyptian Cuisine,Koshary and Basbouussa.

Koshary

Koshary is one of the popular street foods in Egypt,its savored like the Chole chaat in India and Burger in USA.The layers of pasta,rice and lentils are topped with spicy tomato sauce,chickpeas and the crispy fried onions.This is complete vegan meal,the flavors are simple and spicy yet unforgettably delicious.

There is little one can go wrong with the easy recipe.I like the spice from the chili and sweet crispy bits of onion and with lentils,rice and pasta all in one ,the meal was thoroughly fulfilling.

Koshary Recipe
Ingredients
1 cup brown lentils
1 cup rice (I used brown and wild rice blend)
1/2 pound whole grain pasta
1 1/2 teaspoon Salt

For the fried onions :
2 medium onions,sliced
2 tablespoon vegetable oil or extra virgin olive oil

For Spicy tomato sauce(salsa)
2 cups tomato puree or 1 can(8 oz) crushed tomatoes
1/2 a medium onion,finely chopped.
3 garlic cloves, finely chopped.
1 teaspoon hot chili powder
1 teaspoon ground cumin
1/4 teaspoon ground pepper
1 Tablespoon white vinegar
1 tablespoon vegetable oil or extra virgin olive oil

Method
Cook lentils ,pasta and rice
Cook all the 3 separately in salted water.
Cook the rice with 2 cups of water and some salt until tender but not mushy.set aside.
Cook the pasta in salted water until al dente,drain and set aside.
Cook the lentil in 2cups of salted water until tender about 30 minutes in a covered skillet and 9-10 minutes in a pressure cooker on medium heat.drain any excess water.

Fry the Onions
Heat the oil in a large pan,add the onions and saute until nicely brown,stir occasionally.When done transfer on to a paper towel to strain excess oil.
Prepare the tomato sauce
In the same pan ,Saute onions until soft, then add garlic and fry to a pale brown. Add tomato puree,chili powder,cumin and simmer for 15-20 minutes, until sauce is cooked and becomes thick . Add vinegar ,pepper and salt, cook 2-3 more minutes .
To serve
Arrange a layer of cooked pasta,lentils and rice on each of the serving plates.Top with tomato sauce and garnish with fried onions and cooked chickpeas .Serve with a bowl full of Dakka on side.


Suggested Accompaniment - Dakka(Vinegar cumin garlic sauce)
Ingredients needed:
2 teaspoon cumin seeds ,coarsely ground
4 garlic cloves
1/2 a cup of vinegar
1/2 a cup of hot water
1 teaspoon of hot chili
salt and pepper

To make Dakka :
Take garlic cloves and hit them with the end of the knife, but don't break them up. Add into white vinegar and water mix. Add some cumin and coriander into the vinegar as well as salt and pepper and hot chili. Stir and let sit for an hour.Serve on side with Koshary.





Basbouussa(Semolina Cake)

Most celebrated dessert of Egypt,is also famous in other Arab countries.The following is Chahira's Egyptian Basbouussa with Semolina,coconut and yogurt.I made few changes in the sugar and fat quantities,my usual fiddling to make a dessert lower in fat and sugar(Hope you don't mind chahira).
I always spotted this dessert in the middle eastern stores,is almost as popular as baklava.The tantalizing dessert had nice and moist texture,I was pleased to be able to bake one fresh at home.


Recipe
Ingredients
2 cups fine Semolina(sooji) or semolina flour
1 tablespoon baking powder.
1/2 cup dry coconut
3 tablespoon pure ghee(vegan substitute :Virgin pressed Coconut oil)
1 tablespoon Orange blossom water or rose water or 1 teaspoon pure vanilla
1 cup fat free yogurt.(vegan substitute :Soy yogurt)
1/4 cup white or brown sugar(or use half agave nectar & half sugar)

For lite sugar syrup
1/2 cup white or brown sugar*(or use half agave nectar & half sugar)
1 cup of water
1 teaspoon lemon juice.
1 tablespoon orange blossom water or rose water

Tahini and some butter to grease the baking tray.
1/2 cup whole or chopped nuts for garnish(I used almonds)

*for heavy syrup use equal quantity of water and sugar.

Method
In a large bowl,mix the semolina, baking powder, coconut, sugar, ghee, yogurt and blossom water until smooth and well incorporated.
Grease the baking dish little bit of butter(or coconut oil) and tahini, you can use just Tahini.Pour your basbouusa dough and spread it evenly .Garnish it choicest dry fruits and nuts,insert them partially in to the mixture.Set the tray aside for at least an hour.

Bake
Preheat oven to 350F.place the dish in the middle rack of the oven and bake for 35-40 minutes until the top is golden brown.


Prepare the syrup
While the basbousa bakes,prepare the syrup.Boil the sugar,blossom water and water in a small saucepan,until bubbly and in thread point stage.Pour the warm syrup over the warm basbousa.let the flavors soak in for an hour at least.Then slice in squares and serve.



Thank you Chahira for the delicious picks.And Thank you Arlette for making me a part of this exciting club.To join the creative team ,write to Arlette(phoeniciangourmet@sympatico.ca).

Simon Singh - Beware the Spinal Trap

This is the article by Simon Singh, which resulted in a libel lawsuit by the British Chiropractic industry. The alledged libelous comments are in bold....

Beware the spinal trap

Some practitioners claim it is a cure-all but research suggests chiropractic therapy can be lethal

Simon Singh
The Guardian, Saturday April 19 2008

This is Chiropractic Awareness Week. So let's be aware. How about some awareness that may prevent harm and help you make truly informed choices? First, you might be surprised to know that the founder of chiropractic therapy, Daniel David Palmer, wrote that, "99% of all diseases are caused by displaced vertebrae". In the 1860s, Palmer began to develop his theory that the spine was involved in almost every illness because the spinal cord connects the brain to the rest of the body. Therefore any misalignment could cause a problem in distant parts of the body.

In fact, Palmer's first chiropractic intervention supposedly cured a man who had been profoundly deaf for 17 years. His second treatment was equally strange, because he claimed that he treated a patient with heart trouble by correcting a displaced vertebra.

You might think that modern chiropractors restrict themselves to treating back problems, but in fact they still possess some quite wacky ideas. The fundamentalists argue that they can cure anything. And even the more moderate chiropractors have ideas above their station. The British Chiropractic Association claims that their members can help treat children with colic, sleeping and feeding problems, frequent ear infections, asthma and prolonged crying, even though there is not a jot of evidence. This organisation is the respectable face of the chiropractic profession and yet it happily promotes bogus treatments.

I can confidently label these treatments as bogus [changed to "utter nonsense" in the scrubbed version] because I have co-authored a book about alternative medicine with the world's first professor of complementary medicine, Edzard Ernst. He learned chiropractic techniques himself and used them as a doctor. This is when he began to see the need for some critical evaluation. Among other projects, he examined the evidence from 70 trials exploring the benefits of chiropractic therapy in conditions unrelated to the back. He found no evidence to suggest that chiropractors could treat any such conditions.

But what about chiropractic in the context of treating back problems? Manipulating the spine can cure some problems, but results are mixed. To be fair, conventional approaches, such as physiotherapy, also struggle to treat back problems with any consistency. Nevertheless, conventional therapy is still preferable because of the serious dangers associated with chiropractic.

In 2001, a systematic review of five studies revealed that roughly half of all chiropractic patients experience temporary adverse effects, such as pain, numbness, stiffness, dizziness and headaches. These are relatively minor effects, but the frequency is very high, and this has to be weighed against the limited benefit offered by chiropractors.

More worryingly, the hallmark technique of the chiropractor, known as high-velocity, low-amplitude thrust, carries much more significant risks. This involves pushing joints beyond their natural range of motion by applying a short, sharp force. Although this is a safe procedure for most patients, others can suffer dislocations and fractures.

Worse still, manipulation of the neck can damage the vertebral arteries, which supply blood to the brain. So-called vertebral dissection can ultimately cut off the blood supply, which in turn can lead to a stroke and even death. Because there is usually a delay between the vertebral dissection and the blockage of blood to the brain, the link between chiropractic and strokes went unnoticed for many years. Recently, however, it has been possible to identify cases where spinal manipulation has certainly been the cause of vertebral dissection.

Laurie Mathiason was a 20-year-old Canadian waitress who visited a chiropractor 21 times between 1997 and 1998 to relieve her low-back pain. On her penultimate visit she complained of stiffness in her neck. That evening she began dropping plates at the restaurant, so she returned to the chiropractor. As the chiropractor manipulated her neck, Mathiason began to cry, her eyes started to roll, she foamed at the mouth and her body began to convulse. She was rushed to hospital, slipped into a coma and died three days later. At the inquest, the coroner declared: "Laurie died of a ruptured vertebral artery, which occurred in association with a chiropractic manipulation of the neck."

This case is not unique. In Canada alone there have been several other women who have died after receiving chiropractic therapy, and Professor Ernst has identified about 700 cases of serious complications among the medical literature. This should be a major concern for health officials, particularly as under-reporting will mean that the actual number of cases is much higher.

Bearing all of this in mind, I will leave you with one message for Chiropractic Awareness Week - if spinal manipulation were a drug with such serious adverse effects and so little demonstrable benefit, then it would almost certainly have been taken off the market.

· Simon Singh is the co-author of Trick or Treatment? Alternative Medicine on Trial

FitA-Liv, Untuk Kesehatan Hati (Liver) serta Maag Anda

FitA-Liv adalah minuman kesehatan alami berupa serbuk instant siap saji yang terbuat dari ekstrak Curcuma xanthorriza (temulawak), Curcuma domestica (kunyit) dan Crude Centella asiatica (antanan).Di dalam tubuh FitA-Liv akan bekerja di 3 tempat sekaligus yaitu :Di lambung; kandungan kurkumin dalam FitA-Liv yang bersifat basa akan menetralisir kelebihan ASAM LAMBUNG.http://taraprimamegah.com/2009/

FitA-Liv, Untuk Kesehatan Hati (Liver) serta Maag Anda

FitA-Liv adalah minuman kesehatan alami berupa serbuk instant siap saji yang terbuat dari ekstrak Curcuma xanthorriza (temulawak), Curcuma domestica (kunyit) dan Crude Centella asiatica (antanan).Di dalam tubuh FitA-Liv akan bekerja di 3 tempat sekaligus yaitu :Di lambung; kandungan kurkumin dalam FitA-Liv yang bersifat basa akan menetralisir kelebihan ASAM LAMBUNG.http://taraprimamegah.com/2009/

Sunlive - Pills lab goes big

He didn’t set out to clean up the natural health industry but Dr Shaun Holt’s vitamin business has succeeded and is now nominated for a major pharmaceutical company’s award.

He says there’s no irony in a vitamin seller being nominated for an award sponsored by a pharmaceutical company. Bayer is the company that originally marketed aspirin, a derivative of a willow bark natural remedy.

Read more here...

Herbs for health

We are currently trying to grow Goji berries in our garden. I like an Alara muesli that has dry goji berries in it, but it would be fantastic to be able to pick fresh ones and add them to my breakfast muesli.

I've just come across an article which lists 50 herbs and gives some indication about how to grow them. This is what it says about Goji berries:

These all function berries can help the liver, improve fertility, and help you live longer. They grow best in temperate regions.

Not sure about improving fertility (I'm 61), but living longer and helping my liver - definitely.

The Diet-Heart Hypothesis: Subdividing Lipoproteins

Two posts ago, we made the rounds of the commonly measured blood lipids (total cholesterol, LDL, HDL, triglycerides) and how they associate with cardiac risk. It's important to keep in mind that many things associate with cardiac risk, not just blood lipids. For example, men with low serum vitamin D are at a 2.4-fold greater risk of heart attack than men with higher D levels. That alone is roughly equivalent to the predictive power of the blood lipids you get measured at the doctor's office. Coronary calcium scans (a measure of blood vessel calcification) also associate with cardiac risk better than the most commonly measured blood lipids.

Lipoproteins Can be Subdivided into Several Subcategories

In the continual search for better measures of cardiac risk, researchers in the 1980s decided to break down lipoprotein particles into sub-categories. One of these researchers is Dr. Ronald M. Krauss. Krauss published extensively on the association between lipoprotein size and cardiac risk, eventually concluding (source):

The plasma lipoprotein profile accompanying a preponderance of small, dense LDL particles (specifically LDL-III) is associated with up to a threefold increase in the susceptibility of developing [coronary artery disease]. This has been demonstrated in case-control studies of myocardial infarction and angiographically documented coronary disease.

Krauss found that small, dense LDL (sdLDL) doesn't travel alone: it typically comes along with low HDL and high triglycerides*. He called this combination of factors "lipoprotein pattern B"; its opposite is "lipoprotein pattern A": large, buoyant LDL, high HDL and low triglycerides. Incidentally, low HDL and high triglycerides are hallmarks of the metabolic syndrome, the quintessential modern metabolic disorder.

Krauss and his colleagues went on to hypothesize that sdLDL promotes atherosclerosis because of its ability to penetrate the artery wall more easily than large LDL. He and others subsequently showed that sdLDL are also more prone to oxidation than large LDL (1, 2).

Diet Affects LDL Subcategories

The next step in Krauss's research was to see how diet affects lipoprotein patterns. In 1994, he published a study comparing the effects of a low-fat (24%), high-carbohydrate (56%) diet to a "high-fat" (46%), "low-carbohydrate" (34%) diet on lipoprotein patterns. The high-fat diet also happened to be high in saturated fat-- 18% of calories. He found that (quote source):

Out of the 87 men with pattern A on the high-fat diet, 36 converted to pattern B on the low-fat diet... Taken together, these results indicate that in the majority of men, the reduction in LDL cholesterol seen on a low-fat, high-carbohydrate diet is mainly because of a shift from larger, more cholesterol-enriched LDL to smaller, cholesterol-depleted LDL [sdLDL].

In other words, in the majority of people, high-carbohydrate diets lower LDL cholesterol not by decreasing LDL particle count (which might be good), but by decreasing LDL size and increasing sdLDL (probably not good). This has been shown repeatedly, including with a 10% fat diet and in children. However, in people who already exhibit pattern B, reducing fat does reduce LDL particle number. Keep in mind that the majority of carbohydrate in modern America comes from wheat and sugar.

Krauss then specifically explored the effect of saturated fat on LDL size (free full text). He re-analyzed the data from the study above, and found that:

In summary, the present study showed that changes in dietary saturated fat are associated with changes in LDL subclasses in healthy men. An increase in saturated fat, and in particular, myristic acid [as well as palmitic acid], was associated with increases in larger LDL particles (and decreases in smaller LDL particles). LDL particle diameter and peak flotation rate [density] were also positively associated with saturated fat, indicating shifts in LDL-particle distribution toward larger, cholesterol-enriched LDL.

Participants who ate the most saturated fat had the largest LDL, and vice versa. Kudos to Dr. Krauss for publishing these provocative data. It's not an isolated finding. He noted in 1994 that:

Cross-sectional population analyses have suggested an association between reduced LDL particle size and relatively reduced dietary animal-fat intake, and increased consumption of carbohydrates.

Diet Affects HDL Subcategories

Krauss also tested the effect of his dietary intervention on HDL. Several studies have found that the largest HDL particles, HDL2b, associate most strongly with HDL's protective effects (more HDL2b = fewer heart attacks). Compared to the diet high in total fat and saturated fat, the low-fat diet decreased HDL2b significantly. A separate study found that the effect persists at one year. Berglund et al. independently confirmed the finding using the low-fat American Heart Association diet in men and women of diverse racial backgrounds. Here's what they had to say about it:

The results indicate that dietary changes suggested to be prudent for a large segment of the population will primarily affect [i.e., reduce] the concentrations of the most prominent antiatherogenic [anti-heart attack] HDL subpopulation.

Saturated and omega-3 fats selectively increase large HDL. Dr. B. G. of Animal Pharm has written about this a number of times.

Wrapping it Up

Contrary to the simplistic idea that saturated fat increases LDL and thus cardiac risk, total fat and saturated fat have a complex influence on blood lipids, the net effect of which is unclear, but is associated with a lower risk of heart attacks. These blood lipid changes persist for at least one year, so they may represent a long-term effect. It's important to remember that the primary sources of carbohydrate in the modern Western diet are wheat and sugar. Are the blood lipid patterns that associate with heart attack risk in Western countries partially acting as markers of wheat and sugar intake?

* This is why you may read that small, dense LDL is not an "independent predictor" of heart attack risk. Since it travels along with a particular pattern of HDL and triglycerides, in most studies it does not give information on cardiac risk beyond what you can get by measuring other lipoproteins.

The Diet-Heart Hypothesis: Subdividing Lipoproteins

Two posts ago, we made the rounds of the commonly measured blood lipids (total cholesterol, LDL, HDL, triglycerides) and how they associate with cardiac risk. It's important to keep in mind that many things associate with cardiac risk, not just blood lipids. For example, men with low serum vitamin D are at a 2.4-fold greater risk of heart attack than men with higher D levels. That alone is roughly equivalent to the predictive power of the blood lipids you get measured at the doctor's office. Coronary calcium scans (a measure of blood vessel calcification) also associate with cardiac risk better than the most commonly measured blood lipids.

Lipoproteins Can be Subdivided into Several Subcategories

In the continual search for better measures of cardiac risk, researchers in the 1980s decided to break down lipoprotein particles into sub-categories. One of these researchers is Dr. Ronald M. Krauss. Krauss published extensively on the association between lipoprotein size and cardiac risk, eventually concluding (source):

The plasma lipoprotein profile accompanying a preponderance of small, dense LDL particles (specifically LDL-III) is associated with up to a threefold increase in the susceptibility of developing [coronary artery disease]. This has been demonstrated in case-control studies of myocardial infarction and angiographically documented coronary disease.

Krauss found that small, dense LDL (sdLDL) doesn't travel alone: it typically comes along with low HDL and high triglycerides*. He called this combination of factors "lipoprotein pattern B"; its opposite is "lipoprotein pattern A": large, buoyant LDL, high HDL and low triglycerides. Incidentally, low HDL and high triglycerides are hallmarks of the metabolic syndrome, the quintessential modern metabolic disorder.

Krauss and his colleagues went on to hypothesize that sdLDL promotes atherosclerosis because of its ability to penetrate the artery wall more easily than large LDL. He and others subsequently showed that sdLDL are also more prone to oxidation than large LDL (1, 2).

Diet Affects LDL Subcategories

The next step in Krauss's research was to see how diet affects lipoprotein patterns. In 1994, he published a study comparing the effects of a low-fat (24%), high-carbohydrate (56%) diet to a "high-fat" (46%), "low-carbohydrate" (34%) diet on lipoprotein patterns. The high-fat diet also happened to be high in saturated fat-- 18% of calories. He found that (quote source):

Out of the 87 men with pattern A on the high-fat diet, 36 converted to pattern B on the low-fat diet... Taken together, these results indicate that in the majority of men, the reduction in LDL cholesterol seen on a low-fat, high-carbohydrate diet is mainly because of a shift from larger, more cholesterol-enriched LDL to smaller, cholesterol-depleted LDL [sdLDL].

In other words, in the majority of people, high-carbohydrate diets lower LDL cholesterol not by decreasing LDL particle count (which might be good), but by decreasing LDL size and increasing sdLDL (probably not good). This has been shown repeatedly, including with a 10% fat diet and in children. However, in people who already exhibit pattern B, reducing fat does reduce LDL particle number. Keep in mind that the majority of carbohydrate in modern America comes from wheat and sugar.

Krauss then specifically explored the effect of saturated fat on LDL size (free full text). He re-analyzed the data from the study above, and found that:

In summary, the present study showed that changes in dietary saturated fat are associated with changes in LDL subclasses in healthy men. An increase in saturated fat, and in particular, myristic acid [as well as palmitic acid], was associated with increases in larger LDL particles (and decreases in smaller LDL particles). LDL particle diameter and peak flotation rate [density] were also positively associated with saturated fat, indicating shifts in LDL-particle distribution toward larger, cholesterol-enriched LDL.

Participants who ate the most saturated fat had the largest LDL, and vice versa. Kudos to Dr. Krauss for publishing these provocative data. It's not an isolated finding. He noted in 1994 that:

Cross-sectional population analyses have suggested an association between reduced LDL particle size and relatively reduced dietary animal-fat intake, and increased consumption of carbohydrates.

Diet Affects HDL Subcategories

Krauss also tested the effect of his dietary intervention on HDL. Several studies have found that the largest HDL particles, HDL2b, associate most strongly with HDL's protective effects (more HDL2b = fewer heart attacks). Compared to the diet high in total fat and saturated fat, the low-fat diet decreased HDL2b significantly. A separate study found that the effect persists at one year. Berglund et al. independently confirmed the finding using the low-fat American Heart Association diet in men and women of diverse racial backgrounds. Here's what they had to say about it:

The results indicate that dietary changes suggested to be prudent for a large segment of the population will primarily affect [i.e., reduce] the concentrations of the most prominent antiatherogenic [anti-heart attack] HDL subpopulation.

Saturated and omega-3 fats selectively increase large HDL. Dr. B. G. of Animal Pharm has written about this a number of times.

Wrapping it Up

Contrary to the simplistic idea that saturated fat increases LDL and thus cardiac risk, total fat and saturated fat have a complex influence on blood lipids, the net effect of which is unclear, but is associated with a lower risk of heart attacks. These blood lipid changes persist for at least one year, so they may represent a long-term effect. It's important to remember that the primary sources of carbohydrate in the modern Western diet are wheat and sugar. Are the blood lipid patterns that associate with heart attack risk in Western countries partially acting as markers of wheat and sugar intake?

* This is why you may read that small, dense LDL is not an "independent predictor" of heart attack risk. Since it travels along with a particular pattern of HDL and triglycerides, in most studies it does not give information on cardiac risk beyond what you can get by measuring other lipoproteins.

The Diet-Heart Hypothesis: Subdividing Lipoproteins

Two posts ago, we made the rounds of the commonly measured blood lipids (total cholesterol, LDL, HDL, triglycerides) and how they associate with cardiac risk. It's important to keep in mind that many things associate with cardiac risk, not just blood lipids. For example, men with low serum vitamin D are at a 2.4-fold greater risk of heart attack than men with higher D levels. That alone is roughly equivalent to the predictive power of the blood lipids you get measured at the doctor's office. Coronary calcium scans (a measure of blood vessel calcification) also associate with cardiac risk better than the most commonly measured blood lipids.

Lipoproteins Can be Subdivided into Several Subcategories

In the continual search for better measures of cardiac risk, researchers in the 1980s decided to break down lipoprotein particles into sub-categories. One of these researchers is Dr. Ronald M. Krauss. Krauss published extensively on the association between lipoprotein size and cardiac risk, eventually concluding (source):

The plasma lipoprotein profile accompanying a preponderance of small, dense LDL particles (specifically LDL-III) is associated with up to a threefold increase in the susceptibility of developing [coronary artery disease]. This has been demonstrated in case-control studies of myocardial infarction and angiographically documented coronary disease.

Krauss found that small, dense LDL (sdLDL) doesn't travel alone: it typically comes along with low HDL and high triglycerides*. He called this combination of factors "lipoprotein pattern B"; its opposite is "lipoprotein pattern A": large, buoyant LDL, high HDL and low triglycerides. Incidentally, low HDL and high triglycerides are hallmarks of the metabolic syndrome, the quintessential modern metabolic disorder.

Krauss and his colleagues went on to hypothesize that sdLDL promotes atherosclerosis because of its ability to penetrate the artery wall more easily than large LDL. He and others subsequently showed that sdLDL are also more prone to oxidation than large LDL (1, 2).

Diet Affects LDL Subcategories

The next step in Krauss's research was to see how diet affects lipoprotein patterns. In 1994, he published a study comparing the effects of a low-fat (24%), high-carbohydrate (56%) diet to a "high-fat" (46%), "low-carbohydrate" (34%) diet on lipoprotein patterns. The high-fat diet also happened to be high in saturated fat-- 18% of calories. He found that (quote source):

Out of the 87 men with pattern A on the high-fat diet, 36 converted to pattern B on the low-fat diet... Taken together, these results indicate that in the majority of men, the reduction in LDL cholesterol seen on a low-fat, high-carbohydrate diet is mainly because of a shift from larger, more cholesterol-enriched LDL to smaller, cholesterol-depleted LDL [sdLDL].

In other words, in the majority of people, high-carbohydrate diets lower LDL cholesterol not by decreasing LDL particle count (which might be good), but by decreasing LDL size and increasing sdLDL (probably not good). This has been shown repeatedly, including with a 10% fat diet and in children. However, in people who already exhibit pattern B, reducing fat does reduce LDL particle number. Keep in mind that the majority of carbohydrate in modern America comes from wheat and sugar.

Krauss then specifically explored the effect of saturated fat on LDL size (free full text). He re-analyzed the data from the study above, and found that:

In summary, the present study showed that changes in dietary saturated fat are associated with changes in LDL subclasses in healthy men. An increase in saturated fat, and in particular, myristic acid [as well as palmitic acid], was associated with increases in larger LDL particles (and decreases in smaller LDL particles). LDL particle diameter and peak flotation rate [density] were also positively associated with saturated fat, indicating shifts in LDL-particle distribution toward larger, cholesterol-enriched LDL.

Participants who ate the most saturated fat had the largest LDL, and vice versa. Kudos to Dr. Krauss for publishing these provocative data. It's not an isolated finding. He noted in 1994 that:

Cross-sectional population analyses have suggested an association between reduced LDL particle size and relatively reduced dietary animal-fat intake, and increased consumption of carbohydrates.

Diet Affects HDL Subcategories

Krauss also tested the effect of his dietary intervention on HDL. Several studies have found that the largest HDL particles, HDL2b, associate most strongly with HDL's protective effects (more HDL2b = fewer heart attacks). Compared to the diet high in total fat and saturated fat, the low-fat diet decreased HDL2b significantly. A separate study found that the effect persists at one year. Berglund et al. independently confirmed the finding using the low-fat American Heart Association diet in men and women of diverse racial backgrounds. Here's what they had to say about it:

The results indicate that dietary changes suggested to be prudent for a large segment of the population will primarily affect [i.e., reduce] the concentrations of the most prominent antiatherogenic [anti-heart attack] HDL subpopulation.

Saturated and omega-3 fats selectively increase large HDL. Dr. B. G. of Animal Pharm has written about this a number of times.

Wrapping it Up

Contrary to the simplistic idea that saturated fat increases LDL and thus cardiac risk, total fat and saturated fat have a complex influence on blood lipids, the net effect of which is unclear, but is associated with a lower risk of heart attacks. These blood lipid changes persist for at least one year, so they may represent a long-term effect. It's important to remember that the primary sources of carbohydrate in the modern Western diet are wheat and sugar. Are the blood lipid patterns that associate with heart attack risk in Western countries partially acting as markers of wheat and sugar intake?

* This is why you may read that small, dense LDL is not an "independent predictor" of heart attack risk. Since it travels along with a particular pattern of HDL and triglycerides, in most studies it does not give information on cardiac risk beyond what you can get by measuring other lipoproteins.

The Diet-Heart Hypothesis: Subdividing Lipoproteins

Two posts ago, we made the rounds of the commonly measured blood lipids (total cholesterol, LDL, HDL, triglycerides) and how they associate with cardiac risk. It's important to keep in mind that many things associate with cardiac risk, not just blood lipids. For example, men with low serum vitamin D are at a 2.4-fold greater risk of heart attack than men with higher D levels. That alone is roughly equivalent to the predictive power of the blood lipids you get measured at the doctor's office. Coronary calcium scans (a measure of blood vessel calcification) also associate with cardiac risk better than the most commonly measured blood lipids.

Lipoproteins Can be Subdivided into Several Subcategories

In the continual search for better measures of cardiac risk, researchers in the 1980s decided to break down lipoprotein particles into sub-categories. One of these researchers is Dr. Ronald M. Krauss. Krauss published extensively on the association between lipoprotein size and cardiac risk, eventually concluding (source):

The plasma lipoprotein profile accompanying a preponderance of small, dense LDL particles (specifically LDL-III) is associated with up to a threefold increase in the susceptibility of developing [coronary artery disease]. This has been demonstrated in case-control studies of myocardial infarction and angiographically documented coronary disease.

Krauss found that small, dense LDL (sdLDL) doesn't travel alone: it typically comes along with low HDL and high triglycerides*. He called this combination of factors "lipoprotein pattern B"; its opposite is "lipoprotein pattern A": large, buoyant LDL, high HDL and low triglycerides. Incidentally, low HDL and high triglycerides are hallmarks of the metabolic syndrome, the quintessential modern metabolic disorder.

Krauss and his colleagues went on to hypothesize that sdLDL promotes atherosclerosis because of its ability to penetrate the artery wall more easily than large LDL. He and others subsequently showed that sdLDL are also more prone to oxidation than large LDL (1, 2).

Diet Affects LDL Subcategories

The next step in Krauss's research was to see how diet affects lipoprotein patterns. In 1994, he published a study comparing the effects of a low-fat (24%), high-carbohydrate (56%) diet to a "high-fat" (46%), "low-carbohydrate" (34%) diet on lipoprotein patterns. The high-fat diet also happened to be high in saturated fat-- 18% of calories. He found that (quote source):

Out of the 87 men with pattern A on the high-fat diet, 36 converted to pattern B on the low-fat diet... Taken together, these results indicate that in the majority of men, the reduction in LDL cholesterol seen on a low-fat, high-carbohydrate diet is mainly because of a shift from larger, more cholesterol-enriched LDL to smaller, cholesterol-depleted LDL [sdLDL].

In other words, in the majority of people, high-carbohydrate diets lower LDL cholesterol not by decreasing LDL particle count (which might be good), but by decreasing LDL size and increasing sdLDL (probably not good). This has been shown repeatedly, including with a 10% fat diet and in children. However, in people who already exhibit pattern B, reducing fat does reduce LDL particle number. Keep in mind that the majority of carbohydrate in modern America comes from wheat and sugar.

Krauss then specifically explored the effect of saturated fat on LDL size (free full text). He re-analyzed the data from the study above, and found that:

In summary, the present study showed that changes in dietary saturated fat are associated with changes in LDL subclasses in healthy men. An increase in saturated fat, and in particular, myristic acid [as well as palmitic acid], was associated with increases in larger LDL particles (and decreases in smaller LDL particles). LDL particle diameter and peak flotation rate [density] were also positively associated with saturated fat, indicating shifts in LDL-particle distribution toward larger, cholesterol-enriched LDL.

Participants who ate the most saturated fat had the largest LDL, and vice versa. Kudos to Dr. Krauss for publishing these provocative data. It's not an isolated finding. He noted in 1994 that:

Cross-sectional population analyses have suggested an association between reduced LDL particle size and relatively reduced dietary animal-fat intake, and increased consumption of carbohydrates.

Diet Affects HDL Subcategories

Krauss also tested the effect of his dietary intervention on HDL. Several studies have found that the largest HDL particles, HDL2b, associate most strongly with HDL's protective effects (more HDL2b = fewer heart attacks). Compared to the diet high in total fat and saturated fat, the low-fat diet decreased HDL2b significantly. A separate study found that the effect persists at one year. Berglund et al. independently confirmed the finding using the low-fat American Heart Association diet in men and women of diverse racial backgrounds. Here's what they had to say about it:

The results indicate that dietary changes suggested to be prudent for a large segment of the population will primarily affect [i.e., reduce] the concentrations of the most prominent antiatherogenic [anti-heart attack] HDL subpopulation.

Saturated and omega-3 fats selectively increase large HDL. Dr. B. G. of Animal Pharm has written about this a number of times.

Wrapping it Up

Contrary to the simplistic idea that saturated fat increases LDL and thus cardiac risk, total fat and saturated fat have a complex influence on blood lipids, the net effect of which is unclear, but is associated with a lower risk of heart attacks. These blood lipid changes persist for at least one year, so they may represent a long-term effect. It's important to remember that the primary sources of carbohydrate in the modern Western diet are wheat and sugar. Are the blood lipid patterns that associate with heart attack risk in Western countries partially acting as markers of wheat and sugar intake?

* This is why you may read that small, dense LDL is not an "independent predictor" of heart attack risk. Since it travels along with a particular pattern of HDL and triglycerides, in most studies it does not give information on cardiac risk beyond what you can get by measuring other lipoproteins.

Huge study starting on vitamin D & fish oil

Two of the most popular and promising dietary supplements - vitamin D and fish oil - will be tested in a large, US government-sponsored study to see whether either nutrient can lower a healthy person's risk of getting cancer, heart disease or having a stroke.

"If something as simple as taking a vitamin D pill could help lower these risks and eliminate these health disparities, that would be extraordinarily exciting," said Dr. JoAnn Manson. "Vitamin D and omega-3s have powerful anti-inflammatory effects that may be key factors in preventing many diseases. They may also work through other pathways that influence cancer and cardiovascular risk," Manson said.

Read more here...

Kosambari,a raw vegetable salad(With Carrots,Cucumber and Lentils)

A summery salad with fresh veggies including cool cucumber,carrots and tender soaked moong lentils,Kosambari is a favorite side served along with rice,sambar among other tasty dishes in a full course Udupi meal. With minimum or no cooking required the salad\side is definitely a favorable dish for hot summers.

Recipe adapted from Ramya's Mane Adige
Ingredients
1 cup carrots,grated
1/2 cup Moong lentil,soaked (for 4-6 hrs or over night) or use full grown moong sprouts or any other tender sprouts(I used broccoli sprouts).
1 small cucumber,diced
1/2 cup coconut,fresh or dried
1 tablespoon cilantro,finely chopped
1-2 small green chillies,finely chopped(optional)

for dressing
2 teaspoon extra virgin olive oil or coconut oil [exclude the oil here if tempering the salad]
1/2 teaspoon black pepper,freshly ground
1 tablespoon lemon juice,freshly squeezed
1/2 teaspoon salt

optional for Tadka (tempering)
2 teaspoon vegetable oil or coconut oil
1/2 teaspoon mustard seeds
few curry leaves

3 leaves Romaine lettuce(optional)

Method
Mix all the ingredients in a large salad bowl.Whisk all the dressing ingredients in a small bowl and pour in the salad,toss to mix.The salad in the picture is not tempered to keep it as raw as possible, for more flavor temper the salad.Heat the oil and add the seeds and leaves,when seeds start to splutter.turn off the heat.let cool for a minute or 2 before mixing in the salad.Served on fresh lettuce leaves with rice and curry or sambar or rasam.

Kosambari is my entry for Lakshmi's RCI event,hosting Udupi and Mangalorean Cuisine this month is Sia of Monsoon Spice.

help for baby colic

having a baby with colic is distressing both for the mum and for the baby.

A recent study suggests that bacteria in the gut cause inflammation and the distress associated with colic.

Probiotics are known to increase the number of good bacteria and reduce the number of bad bacteria in the gut, but you need to be careful about giving young children probiotics willy-nilly.

Fortunately among all the other great probiotic formulae we sell there's a probiotic formula specifically for children (over 6 months of age). It comes in sachets and so can be added to food.

It's also great for pregnant mums, helping them to give the right strains of bacteria to their baby as it comes down the birth canal.

Hospital stay points out need for health information technology

A recent stay in Hartford Hospital has highlighted the importance of electronic medical records for my personal health care. My doctor’s office, which is located in Hartford Hospital but is apparently not really part of the Hospital, has electronic medical records but Hartford Hospital does not. When I was admitted to the hospital, my doctor’s office faxed over some general information about me, but not a complete medical history. This meant that I had to review my medical history with a doctor even though it was all in the chart in my doctor’s office. The doctors (I saw different ones over the couple of days I was in the hospital) asked me repetitive questions and questions that could have been better answered by a doctor or my medical chart. What if I accidentally gave them the wrong information or forgot to include important facts? Even if Hartford Hospital does get electronic medical records of their own, it is important for them to be able to access information from different doctor’s offices outside of the hospital.

Before my hospital stay, my doctor had sent me to see a specialist, also located in Hartford Hospital but not part of it, and because they were both located in the Hospital, I assumed the specialist would have access to my electronic medical records from my doctor. They did not. This meant that there was information I had to make sure the specialist knew about my situation so they could give me a thorough checkup. It would have been much easier if the specialist’s office had access to the electronic medical records from my doctor’s office. I worry about the care of patients who aren’t organized, with failing memories or because of their illness cannot answer doctors’ questions.
Jen Ramirez

Avoid Dying Before Your Time

Smoking, high blood pressure and being overweight are the leading preventable risk factors for premature mortality in the United States, according to a new study led by researchers at the Harvard School of Public Health (HSPH), with collaborators from the University of Toronto and the Institute for Health Metrics and Evaluation at the University of Washington. The researchers found that smoking is responsible for 467,000 premature deaths each year, high blood pressure for 395,000, and being overweight for 216,000. The effects of smoking work out to be about one in five deaths in American adults, while high blood pressure is responsible for one in six deaths.

It is the most comprehensive study yet to look at how diet, lifestyle and metabolic risk factors for chronic disease contribute to mortality in the U.S. The study appears in the April 28, 2009 edition of the open-access journal PLoS Medicine.

"The large magnitude of the numbers for many of these risks made us pause," said Goodarz Danaei, a doctoral student at HSPH and the lead author of the study. "To have hundreds of thousands of premature deaths caused by these modifiable risk factors is shocking and should motivate a serious look at whether our public health system has sufficient capacity to implement interventions and whether it is currently focusing on the right set of interventions." Majid Ezzati, associate professor of international health at HSPH, is the study's senior author.

The researchers also found large effects from a series of other preventable dietary and lifestyle risk factors. Below are the numbers of deaths in the U.S. due annually to each of the individual risk factors examined:

Smoking: 467,000
High blood pressure: 395,000
Overweight-obesity: 216,000
Inadequate physical activity and inactivity: 191,000
High blood sugar: 190,000
High LDL cholesterol: 113,000
High dietary salt: 102,000
Low dietary omega-3 fatty acids (seafood): 84,000
High dietary trans fatty acids: 82,000
Alcohol use: 64,000 (alcohol use averted a balance of 26,000 deaths from heart disease, stroke and diabetes, because moderate drinking reduces risk of these diseases. But these deaths were outweighed by 90,000 alcohol-related deaths from traffic and other injuries, violence, cancers and a range of other diseases).
Low intake of fruits and vegetables: 58,000
Low dietary poly-unsaturated fatty acids: 15,000

All of the deaths calculated in the study were considered premature or preventable in that the victims would not have died when they did if they had not been subject to the behaviors or activities linked to their deaths.

The Vitamin Lab TV ad...

Doctor finalist in innovation awards

From the SunLive - the Bay's news first

27 Jul 2009

Tauranga's doctor Shaun Holt has been named a finalist in the Science and Health category of the 2009 Bayer Innovators Awards.

The awards recognise top innovators in the fields of Science and Health, Design and Engineering, Agriculture and Environment, Research and Development; and Information Technology and Communications.
Shaun’s invention – The Vitamin Lab – allows consumers to obtain vitamin and supplement recommendations according to their medical requirements. The recommendations are based on medical research studies.
The awards will be announced in Auckland on August 25.

http://www.sunlive.co.nz/7192a1.page

How to Stop Acne ASAP With Home Remedies

There is a home remedy for just about any health issue you have. This includes pimples and acne. If you suffer from acne and/or pimples, there is a reason for it and you first need to know the cause. There are many guides that can tell you How To Stop Acne Asap With Home Remedies, but only one gives you the whole truth and really helps you to eliminate the problem quickly and easily with things you have at home. This means you do not have to spend a lot of money on over the counter acne products or expensive prescription drugs and topical treatments. If you can stop it naturally, why waste your hard earned money.

Every one is different, so there are many natural methods for stopping acne. It is best to have a guide that gives you more than one way to stop your acne and be free from it forever. If you or someone you know has an acne problem, this information is invaluable and can help you achieve healthy looking, soft glowing skin. Some of the How To Stop Acne Asap With Home Remedies methods include things like green tea, tea tree oil, alcohol, toothpaste (not gel) Aloe Vera and many other herbs and natural treatments you can find right inside your home. Not every cure for acne will work for you, so you need to understand the cause of your acne and your skin type in order to unveil the right remedy for you.

Part of stopping acne begins from within. When you have inner balance, your skin will show it. Some things that cause acne include stress, hormonal changes, diet, lack of exercise and prescription medications. There are so many causes for acne that it is important to know which one(s) apply to you. You will not only find out How To Stop Acne Asap With Home Remedies, but you will discover the secret to ending breakouts, redness and even get rid of scarring. Whether you are a teenager suffering from acne or an adult suffering from acne, you need this information. Discover How To Stop Acne Asap With Home Remedies and regain your confidence.

Alex Leguizamo writes informative articles on various subjects including this topic Stop Acne With Home Remedies Please leave the links intact if you wish to reprint this article. Thanks.

Charter Oak after one year – mixed returns

One year after the implementation of the Governor’s Charter Oak Health Plan, the program is still struggling to make a difference for CT’s 326,000 uninsured as outlined in a Stamford Advocate article yesterday, updated today. Currently there are just under 9,000 members accessing care who may not have any other option. However, a year ago the Governor estimated that there would be 15,000 people in the program by now. Over 20,000 people have been unable to qualify for Charter Oak and the program has had difficulty getting providers to sign up.
Ellen Andrews